{"id":1534,"date":"2022-10-07T07:07:08","date_gmt":"2022-10-07T07:07:08","guid":{"rendered":"https:\/\/isletcellsignal.umh.es\/?page_id=1534"},"modified":"2025-04-10T07:56:02","modified_gmt":"2025-04-10T07:56:02","slug":"equip","status":"publish","type":"page","link":"https:\/\/isletcellsignal.umh.es\/ca\/equip\/","title":{"rendered":"Equip"},"content":{"rendered":"

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Catedr\u00e0tic d’Universitat<\/h5>\n

[\/et_pb_text][et_pb_button button_text=”ANGEL NADAL” button_alignment=”center” module_class=”rv_button” _builder_version=”4.17.4″ custom_button=”on” button_text_size=”16px” button_text_color=”#ffffff” button_bg_color=”#ab1a2d” button_border_color=” #ab1a2d” button_border_radius=”13px” background_layout=”dark” global_colors_info=”{}”][\/et_pb_button][et_pb_text module_id=”reveal” _builder_version=”4.17.4″ text_font=”||||||||” text_text_color=”#000000″ text_font_size=”16px” header_2_font=”|600|||||||” header_2_text_color=”#8c0000″ header_2_font_size=”30px” header_2_line_height=”1.2em” global_colors_info=”{}”]<\/p>\n

Angel Nadal \u00e9s catedr\u00e0tic de Fisiologia i subdirector del IDiBE en la Universitat Miguel Hern\u00e1ndez d’Elx i cap de grup i membre del Comit\u00e9 de Direcci\u00f3 del CIBERDEM. Les seues investigacions han demostrat la relaci\u00f3 entre l’exposici\u00f3 al Bisfenol-A i les alteracions del metabolisme de la glucosa, incloent l’estudi dels esdeveniments moleculars inicials. Aquestos resultats han sigut fonamentals per a establir la relaci\u00f3 entre l’exposici\u00f3 als disruptors endocrins i la diabetis mellitus. El seu inter\u00e9s continua en l’estudi de com el medi ambient afecta a la divisi\u00f3, mort i funci\u00f3 de les c\u00e8l\u00b7lules -\u03b2 i la seua implicaci\u00f3 en el desenvolupament de la diabetis. Ha sigut reconegut amb el premi de la Societat Espanyola de Diabetis al millor investigador jove (2004) i el premi Alberto Sols a la investigaci\u00f3 b\u00e0sica en diabetis (2017). Participa habitualment en la millora de la translaci\u00f3 de la ci\u00e8ncia dels disruptors endocrins a la regulaci\u00f3.
https:\/\/orcid.org\/0000-0003-4178-2152<\/span><\/a><\/span><\/p>\n

\n

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CV: cvnadal.pdf<\/a><\/p>\n

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Professor Titular<\/h5>\n

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Juan Mart\u00ednez-Pinna va estudiar Biologia en la Universitat d’Alacant. Es va doctorar a l’Institut de Neuroci\u00e8ncies-CSIC de la Universitat Miguel Hern\u00e1ndez i va realitzar una estada postdoctoral en el Departament de Fisiologia de la Universitat de Cambridge (Regne Unit), on va estudiar la depend\u00e8ncia de voltatge dels receptors acoblats a prote\u00efnes G. \u00c9s professor de Fisiologia i cap del grup d’investigaci\u00f3 de Fisiologia Neuroendocrina de la Universitat d’Alacant. El seu inter\u00e9s cient\u00edfic es centra en l’estudi de l’efecte dels disruptors endocrins en l’alteraci\u00f3 de la fisiologia dels canals i\u00f2nics en les c\u00e8l\u00b7lules-\u03b2 pancre\u00e0tiques i en les neurones. Els resultats de la seua investigaci\u00f3 en l’\u00faltima d\u00e8cada han demostrat que la disfunci\u00f3 dels canals i\u00f2nics indu\u00efda per l’exposici\u00f3 a disruptors endocrins altera la secreci\u00f3 d’insulina en les c\u00e8l\u00b7lules-\u03b2 pancre\u00e0tiques i el llindar del dolor en les neurones sensorials.
https:\/\/orcid.org\/0000-0003-0777-2049<\/span><\/a><\/span><\/p>\n

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Professora Titular<\/h5>\n

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Esther Fuentes va estudiar Bioqu\u00edmica en la Universitat de Val\u00e8ncia (Espanya) i es va doctorar en el King’s College de Londres (Regne Unit). \u00c9s professora de Nutrici\u00f3 i Bromatologia en la Universitat Miguel Hern\u00e1ndez d’Elx. Compagina el seu inter\u00e9s investigador en el paper dels disruptors endocrins en els trastorns metab\u00f2lics amb el seu c\u00e0rrec de Vicerectora adjunta de Cultura.<\/p>\n

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MARINA<\/strong><\/p>\n

SERRANO-MACI\u00c1<\/strong><\/p>\n

Investigadora Postdoctorada<\/span><\/p>\n

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ROBERTO<\/strong><\/p>\n

SEMPERE-NAVARRO<\/strong><\/p>\n

Estudiant de Doctorat<\/span><\/p>\n

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TANIA<\/strong><\/p>\n

EXP\u00d3SITO-GARC\u00cdA<\/strong><\/p>\n

Estudiant de Doctorat<\/span><\/p>\n

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REINALDO<\/strong><\/p>\n

S. DOS SANTOS<\/strong><\/p>\n

Investigador Postdoctorat<\/p>\n

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BEATRIZ <\/strong><\/p>\n

BONMAT\u00cd<\/strong><\/p>\n

T\u00e8cnic de Laboratori<\/span><\/p>\n

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MAR\u00cdA LUISA<\/strong><\/p>\n

NAVARRO<\/strong><\/p>\n

T\u00e8cnic de Laboratori<\/span><\/p>\n

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SALOME <\/strong><\/p>\n

RAM\u00d3N<\/strong><\/p>\n

T\u00e8cnic de Laboratori<\/span><\/p>\n

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CRISTINA\u00a0<\/strong><\/p>\n

QUEREDA<\/strong><\/p>\n

Investigadora Postdoctorada<\/span><\/p>\n

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IGNACIO<\/strong><\/p>\n

BABILONI-CHUST<\/strong><\/p>\n

Estudiant de Doctorat<\/span><\/p>\n

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Catedr\u00e0tic d’UniversitatAngel Nadal \u00e9s catedr\u00e0tic de Fisiologia i subdirector del IDiBE en la Universitat Miguel Hern\u00e1ndez d’Elx i cap de grup i membre del Comit\u00e9 de Direcci\u00f3 del CIBERDEM. Les seues investigacions han demostrat la relaci\u00f3 entre l’exposici\u00f3 al Bisfenol-A i les alteracions del metabolisme de la glucosa, incloent l’estudi dels esdeveniments moleculars inicials. Aquestos […]<\/p>\n","protected":false},"author":3158,"featured_media":0,"parent":0,"menu_order":100,"comment_status":"closed","ping_status":"closed","template":"","meta":{"_et_pb_use_builder":"on","_et_pb_old_content":"

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\u00c1NGEL NADAL<\/h3>
Professor of Physiology<\/h5>

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Professor of Physiology<\/h5>

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\u00c1ngel Nadal is Professor of Physiology and Deputy Chair of IDiBE at the Miguel Hern\u00e1ndez University of Elche and group leader and member of the Steering Committee of CIBERDEM. His research has demonstrated the relationship between Bisphenol-A exposure and alterations in glucose metabolism, including the study of the initial molecular events. These findings have been seminal in establishing the relationship between Endocrine Disruptors exposure and diabetes mellitus. His interest continues in the study of how the environment affects \u03b2-cell division, death and function and its involvement in the development of diabetes. He has been recognized with the Spanish Diabetes Society award for best young researcher (2004) and the Alberto Sols award for basic research in diabetes (2017). He is regularly involved in improving the translation of EDC science to regulation.<\/span><\/p>

https:\/\/orcid.org\/0000-0003-4178-2152<\/span><\/a><\/p>

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JUAN MART\u00cdNEZ-PINNA<\/h3>
Associate Professor<\/h5>

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Associate Professor<\/h5>

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Juan Mart\u00ednez-Pinna studied Biology at the University of Alicante. He obtained his PhD at the\u00a0<\/span>Institute of Neuroscience<\/span>-CSIC of the Miguel Hern\u00e1ndez University and trained as a postdoctoral fellow at the Department of Physiology in the University of Cambridge (UK), where he studied the voltage dependence of G-protein-coupled receptors. He is Lecturer\u00a0of Physiology and head of the Neuroendocrine Physiology research group at the University of Alicante. His scientific interest is focused on the study of the effect of endocrine disruptors with estrogenic activity on the alteration of ion channel physiology in pancreatic\u00a0\u03b2-cells and neurons. His research results during the last decade have shown that ion channel dysfunction induced by exposure to endocrine disruptors alters insulin secretion in pancreatic\u00a0\u03b2-cells and pain threshold in sensory neurons.<\/span><\/p>

https:\/\/orcid.org\/0000-0003-0777-2049<\/span><\/a><\/p>

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ESTHER FUENTES<\/h3>
Associate Professor<\/h5>

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Associate Professor<\/h5>

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Esther Fuentes studied Biochemistry at the University of Valencia, Spain and received her PhD from King's College London, UK. She is Lecturer of Nutrition and Bromatology at the Miguel Hern\u00e1ndez University of Elche. She combines her research interest in the role of endocrine disruptors in metabolic disorders with her position as Deputy Vice Chancellor for Culture.<\/span><\/p>

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IGNACIO<\/strong><\/p>

BABILONI-CHUST<\/strong><\/p>

PhD Student<\/span><\/p>

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BEATRIZ <\/strong><\/p>

BONMAT\u00cd<\/strong><\/p>

Technician<\/span><\/p>

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REINALDO<\/strong><\/p>

S. DOS SANTOS<\/strong><\/p>

Postdoctoral Researcher<\/p>

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REGLA MAR\u00cdA<\/strong><\/p>

MEDINA-GAL\u00cd<\/strong><\/p>

Postdoctoral Researcher<\/span><\/p>

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MAR\u00cdA LUISA<\/strong><\/p>

NAVARRO<\/strong><\/p>

Technician<\/span><\/p>

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SALOME <\/strong><\/p>

RAM\u00d3N<\/strong><\/p>

Technician<\/span><\/p>

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ROBERTO<\/strong><\/p>

SEMPERE-NAVARRO<\/strong><\/p>

PhD Student<\/span><\/p>

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MOLECULAR RESEARCH<\/h4>

To comprehend the molecular differences between alpha and beta cells that allow the former to survive during diabetes development.<\/p><\/div>

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COMPOUND TESTING<\/h4>

To identify and test compounds with the ability to promote human pancreatic beta cell survival. These drugs would be a promising adjuvant therapy in the treatment of type I diabetes.<\/p><\/div>

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We work to understand how chemical pollutants influence glucose homeostasis. Our ultimate goal is to translate this knowledge into actions to decrease the prevalence of diabetes and related metabolic disorders.<\/span><\/h2>

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Diabetes mellitus occurs when blood glucose is too high. Glucose rises after meals and provides energy to our cells after being absorbed with the help of insulin, a hormone produced and secreted by the \u03b2-cells of the islet of Langerhans located in the pancreas. When \u03b2-cells do not produce enough insulin, or insulin signaling is diminished, excessively high blood glucose levels result. To know more about diabetes mellitus visit <\/span><\/p>

https:\/\/www.idf.org\/aboutdiabetes\/what-is-diabetes.html<\/span><\/a><\/span><\/p>

The world is facing a diabetes epidemic. 422 million people worldwide suffer from the disease and one person dies every 7 seconds from diabetes and its complications. To decrease these numbers, it is imperative to investigate what factors cause diabetes and to understand how they do so. This would allow us to intervene and prevent its onset.<\/span><\/p>

The probability of becoming diabetic includes individual genetic predisposition and environmental factors that trigger the onset of the disease. Genes cannot be modified, but the environment can, and by changing the environment we can prevent or delay diabetes.<\/span><\/p>

We can define the environment as everything but the genome. It encompasses multiple factors, from nutrition to chemical pollutants. There is a group of chemical pollutants that are especially important in the etiology of diabetes, the so-called endocrine disruptors or endocrine disrupting chemicals (EDCs). To know more about EDCs visit https:\/\/www.endocrine.org\/topics\/edc<\/span><\/a>.<\/span><\/p>

EDCs are chemicals that mimic, block, or interfere with hormones in the body's endocrine system. We are exposed to low doses of EDCs on a daily basis. Some EDCs increase the likelihood of diabetes indirectly because they cause obesity, but EDCs also damage \u03b2-cells and cause insulin resistance in different tissues. Thus, many EDCs are diabetogenic in their own right. To know more about EDCs and Diabetes Mellitus visit https:\/\/www.diabetesandenvironment.org\/<\/span><\/a><\/span><\/p>

In our group, we focus on how EDCs modify islet cell function and survival to understand how EDCs influence glucose homeostasis. We translate this knowledge into actions to decrease the prevalence of diabetes and related metabolic disorders.<\/span><\/p>

[\/et_pb_text][\/et_pb_column][\/et_pb_row][et_pb_row column_structure=\"1_2,1_2\" disabled_on=\"on|on|on\" _builder_version=\"4.16\" disabled=\"on\" global_colors_info=\"{}\"][et_pb_column type=\"1_2\" _builder_version=\"4.16\" global_colors_info=\"{}\"][et_pb_image src=\"https:\/\/isletcellsignal.umh.es\/files\/2022\/06\/icono-1.png\" align=\"center\" _builder_version=\"4.16\" global_colors_info=\"{}\"][\/et_pb_image][et_pb_text _builder_version=\"4.16\" text_font=\"|300|||||||\" text_font_size=\"19px\" header_2_font=\"|600|||||||\" header_4_font=\"|600|||||||\" header_4_font_size=\"20px\" global_colors_info=\"{}\"]<\/p>

MOLECULAR RESEARCH<\/h4>

To comprehend the molecular differences between alpha and beta cells that allow the former to survive during diabetes development.<\/p><\/div>

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COMPOUND TESTING<\/h4>

To identify and test compounds with the ability to promote human pancreatic beta cell survival. These drugs would be a promising adjuvant therapy in the treatment of type I diabetes.<\/p><\/div>

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[\/et_pb_text][\/et_pb_column][\/et_pb_row][\/et_pb_section]<\/p>

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